ECG of the Week – 26th May 2021 – Interpretation

You have requested an ECG for a 91 year old lady who has multiple co-morbidities and lives in a nursing home. She has presented short of breath. Her ECG is as below.

Interpretation:

  • Rate: 114
  • Rhythm: Flutter (easily confused as sinus when looking at anterior leads, flutter waves seen pre QRS and in T wave in lead II)
  • Axis: Normal (0-90)
  • Morphology: 1.5mm STE aVR, 1mm STE V1, subtle 0.5mm STE aVL, widespread 1-2mm downsloping STD (seen in leads II, III, aVF, V3-6) more subtle 1/2mm STD in I
  • Intervals: QRS 80 QT 320 QTc 445 (Bazetts)
  • Summary: An interesting ECG!

Discussion:

ST elevation in aVR and global ST depression elsewhere is highly indicative of diffuse subendocardial ischaemia which is seen in a variety of cases where the oxygen supply and demand are mismatched. Thinking of it in this manner the STE in aVR is essentially a reciprocal change to the left leads of I,II, aVL, V4-6 as it is electrically opposite (remember aVR overlies atria only not myocardial wall)

Causes include

  • LMCA or Proximal LAD stenosis
    • Studies have shown that STE of >= 1.5 mm is associated with  up to 70% mortality
  • Triple vessel disease
  • Hypoxia – eg PE
  • Hypotension – eg post ROSC, blood loss

Implications for Management:

This would depend entirely on this ladies history.

Differentials include:

  • LAD/LMCA stenosis
    • If she had presented with clinical symptoms of MI she should be managed as a STEMI equivalent (whilst taking into consideration her co-morbidites and functional capacity given she is a 91 year old NH resident) The main change in management would be withholding ticagrelor / clopidogrel if she was a candidate for CABG (increased risk of major bleeding)
    • You could also consider this diagnosis given she is elderly (especially if there was a Hx of DM) without CP as a silent MI with or without secondary APO
  • Rate related ischaemia
    • less likely given rate is only 116 but in a 91 year old lady this may be possible
    • Seen especially in AVNRT and flutter
    • Mechanism not entirely known ? rate related supply and demand mismatch vs ‘unofficial stress test’ unmasking of CAD
  • Other causes of SOB causing supply and demand subendocardial ischaemia
    • PE
    • LRTI
    • COPD etc
    • APO

With thanks to Dr Fiona Beattie and Dr Prathiba Shenoy for their help with interpretation!

Further Reading

Textbook:

Chan TC, Brady WJ, Harrigan RA, Ornato JP, Rosen P. ECG in Emergency Medicine and Acute Care. Elsevier Mosby 2005.

Online:

ST Elevation in aVR